61. PedLynx Beginning in November of 1996, new diseases will be added and OVERDOSE ACHONDROPLASIAACID/ALKALI INGESTION ACNE VULGARIS ACUTE cerebellar ATAXIA ACUTE http://www.icondata.com/health/pedbase/pedlynx.htm

The Pediatric Database (PEDBASE) contains descriptions of over 550 childhood illnesses and has been on the Internet since November 15, 1995. Beginning in November of 1996, new diseases will be added and revisions made to existing files. Information on each disorder in this Database has been obtained from at least 3 sources including Nelson Textbook of Pediatrics (14th and 15th editions), the Birth Defects Encyclopedia (1990 and 1994 editions) and from at least one other source (journal articles, review articles, textbooks). One may download the entire Database as Shareware . An editable Registered version is also available through the author. AARSKOG SYNDROME AASE SYNDROME ABETALIPOPROTEINEMIA ACATALASEMIA ... ZELLWEGER'S SYNDROME

62. Korean Standard Classification Of Diseases neuropathy(G60.) infantile cerebral palsy(G80.-) metabolic disorders(E70-E90) G11.0Congenital nonprogressive ataxia G11.1 Early-onset cerebellar ataxia Early http://www.nso.go.kr/eng/standards/edis/g11.htm

G11 Hereditary ataxia Exclusion £ºhereditary and idiopathic neuropathy(G60.-) infantile cerebral palsy(G80.-) metabolic disorders(E70-E90) G11.0 Congenital nonprogressive ataxia G11.1 Early-onset cerebellar ataxia Early-onset cerebellar ataxia with essential tremor Early-onset cerebellar ataxia with myoclonus[Hunt's ataxia] Early-onset cerebellar ataxia with retained tendon reflexes Friedreich's ataxia(autosomal recessive) X-linked recessive spinocerebellar ataxia G11.2 Late-onset cerebellar ataxia G11.3 Cerebellar ataxia with defective DNA repair Ataxia telangiectasia[Louis-bar] Exclusion : Cockayne's syndrome(Q87.1) xeroderma pigmentosum(Q82.1) G11.4 Hereditary spastic paraplegia G11.8 Other hereditary ataxias G11.9 Hereditary ataxia, unspecified Hereditary cerebellar ataxia NOS Hereditary cerebellar degeneration Hereditary cerebellar disease Hereditary cerebellar syndrome

63. Korean Standard Classification Of Diseases cerebral arteries, not resulting in cerebral infarction Inclusion embolism ofmiddle, anterior and posterior cerebral arteries, and cerebellar arteries, not http://www.nso.go.kr/eng/standards/edis/i66.htm

I66 Occlusion and stenosis of cerebral arteries, not resulting in cerebral infarction Inclusion £ºembolism of middle, anterior and posterior cerebral arteries, and cerebellar arteries, not resulting in cerebral infarction narrowing of middle, anterior and posterior cerebral arteries, and cerebellar arteries, not resulting in cerebral infarction obstruction of middle, anterior and posterior cerebral arteries, and cerebellar arteries, not resulting in cerebral infarction(complete)(partial) thrombosis of middle, anterior and posterior cerebral arteries, and cerebellar arteries, not resulting in cerebral infarction Exclusion £ºwhen causing cerebral infarction(I63.-) I66.0 Occlusion and stenosis of middle cerebral artery I66.1 Occlusion and stenosis of anterior cerebral artery I66.2 Occlusion and stenosis of posterior cerebral artery I66.3 Occlusion and stenosis of cerebellar arteries I66.4 Occlusion and stenosis of multiple and bilateral cerebral arteries I66.8 Occlusion and stenosis of other cerebellar artery Occlusion and stenosis of perforating arteries I66.9 Occlusion and stenosis of unspecified cerebral artery

64. Diseases Of Brain diseases of Brain. Diagnosis, G/M, Organ, Caption. Immature, G, Brain, Immatureinfant brain. Abscess, G, Brain, Abscess. Abscess, G, Brain, cerebellar Abscess. http://155.37.5.42/NAV/MSBrain.HTM

Diseases of Brain Diagnosis G/M Organ Caption Immature G Brain Immature infant brain Immature G Brain Immature Fetal Brain Agenesis of Corpus Callosum G Brain Agenesis of Corpus Callosum Anencephaly G Brain Anencephaly Anencephaly G Brain Anencephaly Anencephaly G Brain Anencephaly with iniencephaly Anencephaly G Brain Anencephaly Anencephaly G Brain Anencephaly Anencephaly G Brain Anencephaly Arnold Chiari Malformation G Brain Arnold-Chiari Malformation Arnold Chiari Malformation G Brain Arnold-Chiari Malformation Arnold Chiari Malformation G Brain Arnold Chiari Malformation Contusion G Brain Old contusion Encephalocele G Brain Encephalocele Encephalocele G Brain Encephalocele Holoprosencephaly G Brain Semilobar holoprosencephaly Holoprosencephaly G Brain Cyclops / Holoprosencephaly Hydranencephaly G Brain Hydranencephaly Hydrocephalus G Brain Hydrocephalus Hydrocephalus G Brain Hydrocephalus Hydrocephalus G Brain Hydrocephalus Lissencephaly G Brain Lissencephaly Microcephaly G Brain Microcephaly Polymicrogyria G Brain Polymicrogyria Porencephaly G Brain Porencephalic Cyst Tuberous sclerosis G Brain Tuberous Sclerosis Abscess G Brain Abscess Abscess G Brain Cerebellar Abscess Abscess M Brain Abscess Abscess M Brain Abscess Abscess M Brain Abscess Abscess M Brain Tissue surrounding Abscess Meningitis G Brain Meningitis and IVH Meningitis M Brain Meningitis Meningitis M Brain Meningitis

65. ICD-9 Listing Of Diseases Infectious diseases. Labyrinthitis secondary to meningitis cerebellar degenerationParkinson's disease Progressive supranuclear palsy Paraneoplastic cerebellar http://www.icsmedical2.com/DiagnosticGuide/icd9list.htm

66. Alphabetical Listing Of Diseases Bony fistula. Cardiac dysrhythmias. cerebellar degeneration. cerebellar hemorrhage.cerebellar infarction. Panic disorder. Paraneoplastic cerebellar degeneration. http://www.icsmedical2.com/DiagnosticGuide/alphalist.htm

67. NORD - National Organization For Rare Disorders, Inc. Rare Disease Database Read about more than 1,100 rare diseases. Viewsample report •. Aniridia cerebellar Ataxia Mental Deficiency. http://www.rarediseases.org/search/rdbdetail_abstract.html?disname=Aniridia Cere

68. NORD - National Organization For Rare Disorders, Inc. related to cancer. Organizations related to cerebellar Degeneration,Subacute American Autoimmune Related diseases Association, Inc. http://www.rarediseases.org/search/rdbdetail_abstract.html?disname=Cerebellar De

69. CMGS-Other Diseases Caused By Dynamic Mutations/17.12.98 1. Human diseases caused by dynamic mutations, other than HD, FraX cerebellar ataxia(ataxic gait, hesitating, explosive speech, rhythmical oscillation of the http://www.ich.ucl.ac.uk/cmgs/otherdyn.htm

MRCPath Self-help course. Birmingham. 1998-1999. 17/12/98. Dynamic mutations. Question 5. Human diseases caused by dynamic mutations, other than HD, FraX, DM and Friedreich's ataxia (FA) include: Dentatorubral-pallidoluysian atrophy (DRPLA). Spinal and bulbar muscular atrophy (Kennedy's syndrome). Spinocerebellar ataxia (SCA): Type I. Type II. Type III (Macado-Joseph disease). Type VI. Type VII. 2. Incidence. DRPLA Very rare in Japan. 2 to 4 per 1,000,000. Very, very rare in Europe and Americas. SMBA Less than 100 families reported by 1996. SCAs Total incidence of 1 per 100,000. SCA I, SCA II and SCA III make up 40%. 3. Symptoms. DRPLA Myolconus (twitching of a muscle group). Epilepsy. Cerebellar ataxia (ataxic gait, hesitating, explosive speech, rhythmical oscillation of the eyeballs and possibly inflammation of the optic nerve). Choreoathetosis (abnormal movement of the body including slow, writhing, involuntary actions commonly involving the hand and feet). Dementia. Dystonia. DRPLA can exhibit clinical features indistinguishable from those of HD, such as choreoathetosis, dementia and cerebellar ataxia. However, epilepsy is not a clinical feature of HD.

70. Generations 2001: Sporadic Cerebellar Degeneration cerebellum. People can have cerebellar disturbances based on causesother than degenerative or hereditary diseases. These include http://www.ataxia.org/generations/2001/2001summer/gilman.html

Sporadic Cerebellar Degeneration presented by Sid Gilman, MD Ann Arbor, MI I have been asked to discuss the sporadic ataxias and I will begin with the classification of the ataxias. Various people use different classifications. The one that I like to use is: Cerebellar Ataxias Classification Symptomatic cerebellar ataxias Sporadic degenerative ataxias Cerebellar ataxia with GAD-Abs Olivopontocerebellar atrophy Hereditary cerebellar atrophy Dominant (SCA1 - 16, DRPLA) Childhood metabolic disorders with cerebellar ataxia The symptomatic ataxias: A number of diseases, of the rest of the body, can affect the brain and the cerebellum. People can have cerebellar disturbances based on causes other than degenerative or hereditary diseases. These include stroke involving the cerebellum, hemorrhage into the cerebellum, loss of blood flow to the cerebellum causing what is called infarction, and abnormal tangles of blood vessels can affect the cerebellum. These often produce cerebellar injury and usually they begin abruptly and it is not hard to make the diagnosis, particularly, now with good medical imaging which helps a lot in this kind of diagnosis. There are also malformations of various sorts. The most common is called Dandy-Walker malformation, this consists of a congenital abnormality in which people are born with a large cyst replacing much of the cerebellum. Oddly enough, these people can be totally normal until adult life, sometimes in their 20's or 30's and sometimes as old as 50's or 60's before symptoms begin. The symptoms are often cerebellar ataxia that may also consist of spasticity with stiffness of the limbs and difficulty ambulating because of that. The other common malformation is called Arnold Chiari syndrome which consists of a variety of abnormalities of the cerebellar structure, too numerous to go into today. They can also show up in adult life although people are born with the malformations.

71. II: CNS Vascular Diseases II. CNS VASCULAR diseases. c) Long circumferential vessels cerebellumand dorsolateral brainstem. i) Superior cerebellar artery. http://www.borg.labmed.umn.edu/PathClass/5102/neuro/clark2.html

Back Home Next II. CNS VASCULAR DISEASES The brain requires 15-20% of normal cardiac output and consumes more oxygen than any other single organ of the body, making it highly vulnerable to injury from hypoxia/ischemia (reduced oxygen/arrest of blood flow). Cerebrovascular events (strokes) result from 1) occlusive vascular disorders, 2) systemic (global) hypoxia/ischemia, and 3) hemorrhage. ANATOMY OF CNS CIRCULATION A) Direct arterial supply to the brain Internal carotid system (supratentorial) a) Small paramedian vessels - perforate base of brain near the midline b) Short circumferential vessels - arise from proximal major arteries and supply thalamus, internal capsule, and basal ganglia; lenticulostriate and thalamostriate arteries (vulnerable to hypertensive changes) c) Long circumferential vessels - cortex and white matter i) Anterior cerebral artery - has good left-right collaterals ii) Middle cerebral artery - receives ~80% of ICA flow iii) Posterior cerebral artery - functionally a part of the basilar system in most people Vertebral-basilar system (infratentorial - except for PCA) a) Small paramedian vessels - perforate midline ventral brainstem b) Short circumferential vessels - lateral brainstem (vulnerable to hypertensive changes) c) Long circumferential vessels - cerebellum and dorsolateral brainstem i) Superior cerebellar artery ii) Anterior inferior cerebellar artery

72. THE MERCK MANUAL, Sec. 14, Ch. 180, Demyelinating Diseases Chapter 180. Demyelinating diseases. Topics. General. Multiple Sclerosis. Nystagmus,a common finding, may be due to cerebellar or vestibular nucleus damage. http://www.merck.com/pubs/mmanual/section14/chapter180/180b.htm

This Publication Is Searchable The Merck Manual of Diagnosis and Therapy Section 14. Neurologic Disorders Chapter 180. Demyelinating Diseases Topics [General] Multiple Sclerosis Multiple Sclerosis A slowly progressive CNS disease characterized by disseminated patches of demyelination in the brain and spinal cord, resulting in multiple and varied neurologic symptoms and signs, usually with remissions and exacerbations. Etiology and Incidence The cause is unknown, but an immunologic abnormality is suspected. One postulated cause is infection by a latent virus (possibly by a human herpesvirus or retrovirus), in which viral activation and expression trigger a secondary immune response. An increased family incidence and association with certain HLA allotypes suggest genetic susceptibility. Environment may be a factor: MS is more common in temperate climates (1/2000) than in the tropics (1/10,000). It has been linked to the geographic area in which a patient's first 15 yr are spent; relocation after age 15 does not alter the risk. Age at onset is typically 20 to 40 yr, and women are affected somewhat more often than men. Pathology Plaques of demyelination, with destruction of oligodendroglia and perivascular inflammation, are disseminated throughout the CNS, primarily in the white matter, with a predilection for the lateral and posterior columns (especially in the cervical and dorsal regions), the optic nerves, and periventricular areas. Tracts in the midbrain, pons, and cerebellum are also affected as is gray matter in the cerebrum and spinal cord. Cell bodies and axons are usually preserved, especially in recent lesions. Later, axons may be destroyed, especially in the long tracts, and a fibrous gliosis makes the tracts appear sclerotic. Recent and old lesions may coexist. Chemical changes in lipid and protein constituents of myelin occur in and around the plaques.

73. THE MERCK MANUAL, Sec. 13, Ch. 162, Viral Diseases As the diseases progress, certain tests become more diagnostic. A CT brainscan may show cerebral and cerebellar atrophy but is usually normal. http://www.merck.com/pubs/mmanual/section13/chapter162/162d.htm

This Publication Is Searchable The Merck Manual of Diagnosis and Therapy Section 13. Infectious Diseases Chapter 162. Viral Diseases Topics [General] Respiratory Viral Diseases Herpesvirus Infections Central Nervous System Viral Diseases ... Smallpox Central Nervous System Viral Diseases RABIES (Hydrophobia) An acute infectious disease of mammals, especially carnivores, characterized by CNS pathology leading to paralysis and death. Etiology and Epidemiology Rabies is caused by a neurotropic virus often present in the saliva of rabid animals. Isolates of rabies virus collected from different animal species and from different parts of the world are distinct. Rabid animals transmit the infection by biting animals or humans. Rabies is rarely transmitted from infected saliva to a mucous membrane or skin abrasion. Other rare cases of respiratory infections followed exposure in the laboratory and from the atmosphere of a bat-infested cave. Worldwide, rabid dogs still present the highest risk to humans. Rabies in dogs is prevalent in Latin America, Africa, and Asia. In the USA, where vaccination has largely eliminated canine rabies, bites of infected wild animals, especially bats, have caused most of the infrequent cases of human rabies since 1960. Rabid dogs may have furious rabies

74. Rare Diseases List - Office Of Rare Diseases Genetic and Rare diseases Information Center is supported by the Office of Rarediseases in partnership Disease Aniridia cerebellar ataxia mental deficiency. http://ord.aspensys.com/diseaseinfo.asp?ID=13

75. DISEASES OF SPINE diseases OF SPINE. This is classified into. 1 TYPE 1. Where there is herniationof the cerebellar tonsils into the upper cervical canal. 2 TYPE 2. http://www.health.adelaide.edu.au/paed-neuro/diseases_of_spine1.htm

DISEASES OF SPINE Conditions of the spinal column and intervertebral disc will not be discussed as they have little neurosurgical involvement in children. Spinal trauma is uncommon in paediatric practice and will not be discussed. Conditions affecting the paediatric spine are unique. Most of the conditions are congenital but they may persist until older age. Often these conditions involve the spinal cord, spinal nerves as well as the spinal column. I will discuss the common conditions that are seen in Paediatric age group. SYRINGOMYELIA This is a condition where there is central cavitation of the spinal cord. It starts by dilatation of the central canal, as the dilatation progresses it extends into the parenchyma of the spinal cord. At the end there is a very large central cavity filled with clear fluid surrounded by a thin rim of spinal cord. Usually the cavity wall is not smooth but septated. It is surprising that in spite of the extent of the central cavitation and the thin rim of the spinal cord tissue left very little clinical signs are seen. Syringomyelia is classified as a primary and syringomyelia that is secondary to other existing abnormalities.

76. Ataxia & Cerebellar Disorders cerebellar Disorders Intracranial Pressure Neurological Symptoms Signs NeurotoxicologyNeurology Neuroscience Books Autonomic Nervous Ssystem diseases http://www.medlina.com/ataxia_&_cerebellar__disorders.htm

Academic Medicine Addiction Aging AIDS-HIV ... Women's Health The Web MEDLINA.com (partial) CDC WHO FDA NIH CATEGORIES Search: All Products Books Magazines Popular Music Classical Music Video DVD Baby Electronics Software Outdoor Living Wireless Phones Keywords: Home Up Neurology Neuroscience Books Neuroanatomy ... A-T Children's Project - http://www.atcp.org A clearinghouse for ataxia-telangiectasia information, including disease information, scientific research, clinical and family support, and fundraising. National Ataxia Foundation - http://www.ataxia.org Information about and support for people with ataxia. Ataxia Telangiectasia Society - http://www.atsociety.org.uk Details about the organization which is committed to helping, supporting and advising families affected by AT. Includes research, support and awareness. Friedreich's Ataxia Fact Sheet - http://www.ninds.nih.gov A comprehensive resources guide and fact sheet about Friedreich's Ataxia, compiled by NINDS, the National Institute of Neurological Disorders and Stroke. CajunHeart's HomePage - Living Life with Ataxia - http://cajunheart.tripod.com

77. WebGuest - Open Directory : Health : Conditions And Diseases : Neurological Diso Sites International Paraneoplastic Association An article about paraneoplasticcerebellar degeneration with an explanation what it is, diagnosis, treatment http://directory.webguest.com/index.cgi/Health/Conditions_and_Diseases/Neurologi

Browse thru 1000's of books about health, mind and body: About Us Privacy Statement Acceptable Use Policy Legal Notices ... Contact Us the entire directory only in Brain_Diseases/Cerebellar Top Health Conditions and Diseases Neurological Disorders ... Brain Diseases : Cerebellar Ataxia Dandy Walker Syndrome Miller Fisher Syndrome Spinocerebellar Degenerations Sites: International Paraneoplastic Association - An article about paraneoplastic cerebellar degeneration with an explanation what it is, diagnosis, treatment, research and some notes to care givers and family members. The Merck Manual: Disorders Of Movement - Information about cerebellar and spinocerebellar disorders. Includes a short discussion about friedreich's ataxia, cerebellar ataxias and multiple systems atrophy. Paraneoplastic Cerebellar Degeneration - An article written by Timothy C. Hain, MD. WorldMedicus - A description of cerebellar neoplasms along with links to reviews, articles and images. Last update: 13:24 PT, Monday, January 21, 2002 Help build the largest human-edited directory on the web.

78. Diseases - Search atrophy, Familial progressive cerebellar ataxia, Spinal cerebellar atrophy with RehabilitationResearch and Training Center in Neuromuscular diseases (RRTC/NMD http://www.rehabinfo.net/resources/diseases/list/

NIDRR Rehabilitation Research and Training Center in Neuromuscular Diseases (RRTC/NMD) Search Site Path: Home resources diseases list March 30, 2003 [ Sorted by Name ] Sort by Search Term Search for keywords: A B C D ... Acid Maltase Deficiency Glycogenosis type II, Pompe disease, Glycogen storage disease II, Generalized glycogenosis, Lysosomal alpha-glucosidase deficiency, Amyotrophic Lateral Sclerosis Motor neuron disease, Familial amyotrophic lateral sclerosis (FALS), ALS, Lou Gehrig's disease, Becker Muscular Dystrophy (BMD) Benign juvenile muscular dystrophy, Progressive tardive muscular dystrophy, Carnitine Deficiency Systemic carnitine deficiency, Muscle carnitine deficiency, Renal reabsorption of carnitine defect, Central Core Disease (CCD) Muscle core disease, Muscular central core disease, Central fibrillar myopathy, Shy-Magee syndrome, Central core disease of muscle, Central core myopathy, Charcot-Marie-Tooth Disease (CMT) Hereditary sensory motor neuropathy (HSMN), Peroneal muscular atrophy (PMA), Neuropathy with focally folded myelin sheaths, Congenital hypomyelinating neuropathy, Congenital Fiber-type Disproportion (CFTD) Atrophy of Type I fibers, Myopathy of congenital fiber-type disproportion, CFTDM

79. Prion Diseases analysis led to the discovery of further prion diseases including fatal presentsitself with the following clinical features chronic cerebellar ataxia with http://fbspcu01.leeds.ac.uk/users/bmb9aej/Priondiseases.html

80. Nat'l Academies Press, Infectious Diseases Of Mice And Rats (1991), 12. Multiple Infectious diseases of Mice and Rats (1991) Institute for Laboratory Animal virus,KRV infection, wild mice, laboratory mice, cerebellar hypoplasia, virus http://www.nap.edu/books/0309063329/html/236.html

Infectious Diseases of Mice and Rats Institute for Laboratory Animal Research ( ILAR Related Books Openbook Linked Table of Contents Front Matter, pp. i-viii Contents, pp. ix-xii I. Principles of Rodent Disease Pre..., pp. 1-2 1. Objectives, Terminology, and Ove..., pp. 3-12 2. Breeding, Transportation, and Us..., pp. 13-16 3. Barrier Programs, pp. 17-20 4. Health Surveillance Programs, pp. 21-28 II. Individual Disease Agents and T..., pp. 29-30 5. Introduction, pp. 31-32 6. Respiratory System, pp. 33-84 7. Digestive System, pp. 85-163 8. Skin and Joints, pp. 164-197 9. Hemopoietic System, pp. 198-221 10. Central Nervous System, pp. 222-230 11. Genitourinary System, pp. 231-235 12. Multiple Systems, pp. 236-256 III. Indexes to Diagnosis and Resea..., pp. 257-258 Introduction, pp. 259-259 Clinical Signs, pp. 260-265 Pathology, pp. 266-274 Research Complications, pp. 275-276 References, pp. 277-386 Index, pp. 387-397 THIS PAGE You may want to explore these Related Books Openbook Linked Table of Contents Front Matter, pp. i-viii Contents, pp. ix-xii

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