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         Hyperparathyroidism:     more books (71)
  1. The 'sentinel clot' sign in spontaneous retropharyngeal hematoma secondary to parathyroid apoplexy.: An article from: Ear, Nose and Throat Journal by George Koulouris, Marcus Pianta, et all 2006-09-01
  2. Hyperparathyroid-related cognitive dysfunction improves with surgery.(Metabolic Disorders): An article from: Family Practice News by Bruce Jancin, 2007-03-15
  3. Gale Encyclopedia of Medicine: Parathyroid hormone test by Janis O. Flores, 2002-01-01
  4. Postop PTH test unnecessary when calcium is normal.(Metabolic Disorders)(parathyroid hormone): An article from: Family Practice News by Jeff Evans, 2006-05-01
  5. Cinacalcet lowers PTH and calcium in dialysis patients.(parathyroid hormone ): An article from: Internal Medicine News by Steve Perlstein, 2004-07-15
  6. Audio-digest: General Surgery: Management of Thyroid Diseases (Vol. 49, Issue 13, July 7, 2002) by MD Jon A. Heerden, 2002
  7. Cost-effective workup for kidney stones (Postgraduate Medicine) by MD Neil Baum, 2010-06-21
  8. Surgery of the Thyroid and Parathyroid Glands (Clinical Surgery International) (No.6)
  9. The logic of parathyroid surgery and the viscerovertebral angle by Louis T Tenta, 1987

81. Hyperparathyroidism -- Endocrine Resource Center -- Thyroid Disease Site
hyperparathyroidism part of the Endocrine Resource Center, featuring informationon Addison's Disease, Cushing's Syndrome, hyperparathyroidism, Multiple
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Hyperparathyroidism
Primary hyperparathyroidism is a disorder of the parathyroid glands. Most people with this disorder have one or more enlarged, overactive parathyroid glands that secrete too much parathyroid hormone. In secondary hyperparathyroidism, a problem such as kidney failure makes the body resistant to the action of parathyroid hormone. This e-pub focuses on primary hyperparathyroidism.
What Are the Parathyroid Glands?

82. MJA: Edis, Hyperparathyroidism: What Does Surgery Have To Offer?
hyperparathyroidism what does surgery have to offer? Surgery for primaryhyperparathyroidism 19621996 indications and outcomes.
http://www.mja.com.au/public/issues/feb16/edis/edis.html
Home Issues Classifieds More... ... Search
Hyperparathyroidism: what does surgery have to offer?
Even asymptomatic patients should be considered for surgery MJA
The report by Delbridge et al in this issue of the Journal describes the changing clinical picture of primary hyperparathyroidism (PHPT) in patients referred for surgical treatment at Royal North Shore Hospital, Sydney, over the past 30 years. The most common presentation in the 1990s is in women over the age of 50 years with bone loss indistinguishable from that of postmenopausal or senile osteoporosis (31%). Patients with kidney stones still constitute 15%, and those with subtle but distressing symptoms of fatigue, depression or altered cognitive function constitute 20%. Delbridge et al have also observed an "exponential" increase in the number of cases referred for surgery over the past decade, reflecting contemporary epidemiological studies which have unearthed a virtual epidemic of PHPT among postmenopausal women it affects 2% to 3% of women in this age group. Even so, the absolute number of patients undergoing operation at Royal North Shore Hospital each year remains small when compared with the number expected by extrapolation from this prevalence. Presumably, this is due in part to underdiagnosis of PHPT, as well as continuing reluctance among physicians to refer patients for surgery unless they have marked hypercalcaemia or overt "bone or stone" disease.

83. MJA: Delbridge Et Al., Surgery For Primary Hyperparathyroidism 1962-1996: Indica
Surgery for primary hyperparathyroidism 19621996 indications and outcomes. Isparathyroidectomy of benefit in primary hyperparathyroidism?
http://www.mja.com.au/public/issues/feb16/delbridge/delb.html
Home Issues Classifieds More... ... Search
Surgery for primary hyperparathyroidism 1962-1996: indications and outcomes
Leigh W Delbridge, Nidal A Younes, Ana I Guinea,
Thomas S Reeve, Phillip Clifton-Bligh and Bruce G Robinson
MJA
For editorial comment see Edis Abstract Introduction Methods ... Current contents list MJA
Abstract
Objective: To examine changes over the past three decades in the indications for, and outcomes of, surgery for primary hyperparathyroidism.
Design: Survey of a prospective hospital database.
Setting: Royal North Shore Hospital (a tertiary referral and university teaching hospital), Sydney, New South Wales, January 1962 to December 1996.
Patients: All 733 patients who underwent neck exploration for primary hyperparathyroidism.
Results: The annual number of parathyroidectomies increased virtually exponentially, from a mean of two in 1962-1969 to 73 in 1996. In the 1960s and 1970s, the most common indication for surgery was the presence of renal calculi (58% and 43%, respectively), but in the 1980s there was a marked increase in presentation of asymptomatic disease after biochemical screening (19%). In the 1990s, low bone mineral density detected by osteodensitometry has become the most common indication for surgery (31%). After initial operation, 11 patients (2%) had persistent hypercalcaemia, with five of these cured by reoperation an overall failure rate of 1%.
Conclusions: Surgery for primary hyperparathyroidism has become increasingly common, with low bone mineral density replacing renal calculi as the most common indication for surgery. Neck exploration in experienced hands results in an overall cure rate of 99%.

84. Primary Hyperparathyroidism
Parathyroid. Primary hyperparathyroidism. In primary parathyroid hormone.Primary hyperparathyroidism What are the parathyroid glands?
http://jhhs.client.web-health.com/web-health/topics/GeneralHealth/generalhealths

85. Hyperparathyroidism - Overview
hyperparathyroidism. Definition excessive production of parathyroid hormoneby the parathyroid glands. Causes, incidence, and risk factors
http://www.wfubmc.edu/besthealth/ency/article/001215.htm
Disease Injury Nutrition Poison ... Prevention Hyperparathyroidism Definition: excessive production of parathyroid hormone by the parathyroid glands. Causes, incidence, and risk factors: Hyperparathyroidism can be caused by enlargement of one or more of the parathyroid glands or in response to abnormally low calcium levels in the blood.
The parathyroid glands are located at the front and base of the neck around the thyroid gland and produce parathyroid hormone . Parathyroid hormone regulates calcium, phosphorus, and magnesium balance within the blood and bone by maintaining a balance between the mineral levels in the blood and the bone. Excessive levels of parathyroid hormone disturbs this balance and results in increasing the blood calcium level by reabsorbing it from the bone and increasing absorption from the kidneys and intestines.
The effects of abnormal calcium levels are seen in several body systems including the skeletal, gastrointestinal, renal , muscular, and central nervous system
Related topics: Review Date: 6/1/01 Reviewed By: Carlos Bernal-Mizrachi, M.D., Department of Endocrine and Metabolism, Washington University, St. Louis, MO. Review provided by VeriMed Healthcare Network.

86. Diabetes & Other Endocrine And Metabolic Disorders - Hyperparathyroidism
here. hyperparathyroidism. What is hyperparathyroidism? hyperparathyroidismis caused by overactive parathyroid glands. Overactive
http://www.chkd.org/Diabetes/hpt.asp
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For a doctor who specializes in this topic, click here. Hyperparathyroidism What is hyperparathyroidism?
Hyperparathyroidism is caused by overactive parathyroid glands. Overactive parathyroid glands produce high levels of parathyroid hormones, which, in turn, results in increased levels of calcium in the blood stream. The excess calcium released by the bones leads to osteoporosis and osteomalacia (both bone-weakening diseases). Another result of hyperparathyroidism is kidney stones, because of high levels of calcium excreted into the urine by the kidneys. Hyperparathyroidism is quite rare in children. What causes hyperparathyroidism?

87. Hyperparathyroidism - Clinical
First Previous Next Last Index Text. Slide 39 of 64.
http://pathophysiology.uams.edu/Spring01/endo/Thyroid-Parathyroid-Lecture 28/sld
First Previous Next Last ... Text Slide 39 of 64

88. Hyperparathyroidism
hyperparathyroidism. In secondary hyperparathyroidism, a problem such as kidneyfailure makes the body resistant to the action of parathyroid hormone.
http://www.thyroid.ca/Articles/EngE4L.html
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Hyperparathyroidism
Numerous requests have been received for information about hyperparathyrodism. It is hoped that this article may be helpful.. Editor Primary hyperparathyrodism is a disorder of the parathyroid glands. Most people with this disorder have one or more enlarged, overactive parathyroid glands that secrete too much parathyroid hormone. In secondary hyperparathyroidism, a problem such as kidney failure makes the body resistant to the action of parathyroid hormone. This article focuses on primary hyperparathyrodism. What are the parathyroid glands? The parathyroid glands are four pea-sized glands located on the thyroid gland in the neck. Occasionally, a person is born with one or more of the parathyroid glands embedded in the thyroid, the thymus, or elsewhere in the chest. In most such cases, however, the glands function normally. Though their names are similar, the thyroid and parathyroid glands are entirely separate glands, each producing distinct hormones with specific functions. The parathyroid glands secrete parathyroid hormone (PTH), a substance that helps maintain the correct balance of calcium and phosphorus in the body.

89. Hyperparathyroidism From Pediatrics / Endocrinology
hyperparathyroidism hyperparathyroidism describes proliferation of the parathyroidhormone (PTH)-secreting cells, or chief cells, in one or more of the 4
http://author.emedicine.com/ped/topic1086.htm
eMedicine Journal Pediatrics Endocrinology
Hyperparathyroidism
Synonyms, Key Words, and Related Terms: parathyroid adenoma, parathyroid hyperplasia Author Information Introduction Clinical Differentials ... Bibliography
AUTHOR INFORMATION Section 1 of 11 Authored by Gordon L Klein, MD, MPH , Professor, Departments of Pediatric Gastroenterology, Hepatology, and Nutrition, University of Texas Medical Branch Gordon L Klein, MD, MPH, is a member of the following medical societies: American Academy of Pediatrics American Gastroenterological Association American Pediatric Society American Society for Bone and Mineral Research ... Sigma Xi , and Society for Pediatric Research Edited by Phyllis Speiser, MD , Director, Division of Pediatric Endocrinology, Children's Health Network of North Shore/LIJ Health System, Clinical Professor, Department of Pediatrics, New York University School of Medicine; Robert Konop, PharmD , Clinical Assistant Professor, Department of Pharmacy, Section of Clinical Pharmacology, University of Minnesota; Stephen Kemp, MD, PhD

90. Bone Densitometry: Patients With Asymptomatic Primary Hyperparathyroidism
Bone Densitometry Patients with Asymptomatic Primary hyperparathyroidism. TitleBone Densitometry Patients with Asymptomatic Primary hyperparathyroidism.
http://www.ahcpr.gov/clinic/aphtbone.htm
Bone Densitometry: Patients with Asymptomatic Primary Hyperparathyroidism
Title : Bone Densitometry: Patients with Asymptomatic Primary Hyperparathyroidism. Agency : Agency for Health Care Policy and Research/Center for Health Care Technology (formerly the Office of Health Technology Assessment). Contact : Martin Erlichman, M.S., and Thomas V. Holohan, M.D., FACP. Status : Technology Assessment: Published, 1996. Language : English Primary Objective : To assess which techniques, if any, are clinically useful in the medical management of patients with bone loss due to asymptomatic primary hyperparathyroidism. Methods Used : Review of published literature, collection of information from interested public in response to a Federal Register notice of intent to undertake assessment, solicitation of data from other agencies of the U.S. Public Health Service (PHS). Data Identification : Published literature identified by search of the MEDLINE database, references from bibliographies of reviewed articles, data submitted by other Federal agencies. Study Selection : All published material of primary data addressing bone loss in patients with PHPT, fracture risk in patients with PHPT and changes in BMD following surgery for PHPT.

91. PRIMARY HYPERPARATHYROIDISM IN AN INFANT: CASE REPORT WITH REVIEW OF THE LITERAT
May 1995. PRIMARY hyperparathyroidism IN AN INFANT CASE REPORT WITH REVIEWOF THE LITERATURE. Primary hyperparathyroidism is a rare cause.
http://www.kfshrc.edu.sa/annals/153/94133.html
May 1995
PRIMARY HYPERPARATHYROIDISM IN AN INFANT: CASE REPORT WITH REVIEW OF THE LITERATURE
Abdullah Salim Al-Jarallah, MBChB, ABP; Abdullah Solaiman Al-Herbish, MBBS, FRCPC, FAAP; Abdullah Al-Rabeeah, MBBS, FRCSC; Nimer Suliman, MBBS, MRCP From the Department of Pediatrics, King Khalid University Hospital (Drs. Al-Jarallah, Al-Herbish), King Faisal Specialist Hospital and Research Centre (Dr. Al-Rabeeah), and Riyadh Medical Complex (Dr. Suliman), Riyadh. Address reprint requests and correspondence to Dr. Al-Herbish: P.O. Box 90533, Riyadh 11623, Saudi Arabia. Accepted for publication 10 July 1994. Primary hyperparathyroidism is rare in children under the age of sixteen and uncommon in adolescents.1 While most cases of primary hyperparathyroidism in neonates and infants are caused by parathyroid hyperplasia, the majority of cases appearing during childhood and adolescence are caused by a parathyroid adenoma.2 A female Sudanese infant who has primary hyperparathyroidism due to parathyroid hyperplasia is presented. Results of workup, possible etiologies and review of the literature on primary hyperparathyroidism are presented. To our knowledge, this is the first case reported at this age in the Arab world.
Case Report
Laboratory investigations showed calcium level of 6 mmol/L (N = 2.1 to 2.6), phosphate 2.2 mmol/L (N = 0.8 to 1.4), magnesium 1.8 mmol/L, alkaline phosphatase 670, albumin 47 g/L, urine Ca/urine creatinine ratio 9.6 (N = 0.7), vitamin D level 43 (N = 20 to 120), serum parathyroid hormone (PTH) 285 (N = 5 to 45) with calcium level of 4.22, urine homovanillic acid (HVA) 0.3 mg/24 hours (N = up to 4.1). Vitamin D, PHT and HVA were done by Bioscientia Laboratories in Germany. The patient's father, mother and uncle had normal calcium levels. Electrocardiogram showed sinus tachycardia. Echocardiogram showed normal aortic arch. Abdominal and neck ultrasound, x-ray of both hands, and brain and neck computed tomography (CT) scans were also normal. Parathyroid scan (thallium scan) showed a hot area in the right lower parathyroid gland with suspicion of parathyroid adenoma.

92. Primary Hyperparathyroidism
Primary hyperparathyroidism Primary hyperparathyroidism results from disorders intrinsicto the parathyroid glands, and occurs in approximately 1 in 800 adults
http://www.echt.chm.msu.edu/courseware/blockII/Pathology/Parathyroid_4.html
echt home block II pathology parathyroid menu
Primary Hyperparathyroidism
Primary hyperparathyroidism results from disorders intrinsic to the parathyroid glands, and occurs in approximately 1 in 800 adults. The hypersecretion of parathyroid hormone results in hypercalcemia (34% of the clinical causes of hypercalcemia), hypophosphatemia, and excessive urinary excretion of calcium.
The earliest clinical signs are neuromuscular weakness and fatigue and neuropsychiatric disturbances ranging from depression, anxiety and psychoses. Renal stones or nephrocalcinosis due to hypercalciuria are seen in 50 - 70% of patients, and is the most common clinical presentation. Reabsorption and remodeling of bone may result in osteitis fibrosa cystica, which is diagnostic of hyperparathyroidism. Soft tissue calcifications may occur in about 15 - 20 % of the cases, due to hypercalcemia. Remember: not all hypercalcemia is due to hyperparathyroidism. Nonparathyroid malignancy is the most common cause (35%) of hypercalcemia in hospitalized patients. Other causes include vitamin D excess (12%), hyperthyroidism (4%), milk alkali syndrome (4%), and immobilization (2.3%). Laboratory error may account for as much as
13.4% of elevated serum calcium levels. Other conditions with

93. Hyperphosphatemia--- Hyperparathyroidism
First Previous Next Last Index Home Text. Slide 10 of 33.
http://www.wramc.amedd.army.mil/departments/medicine/Nephrology/education/Lectur

94. Diabetes & Other Endocrine And Metabolic Disorders - Hyperparathyroidism
hyperparathyroidism. What is hyperparathyroidism? hyperparathyroidism is causedby overactive parathyroid glands. What causes hyperparathyroidism?
http://www.musckids.com/health_library/diabetes/hpt.htm

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Hyperparathyroidism
What is hyperparathyroidism?
Hyperparathyroidism is caused by overactive parathyroid glands. Overactive parathyroid glands produce high levels of parathyroid hormones, which, in turn, results in increased levels of calcium in the blood stream. The excess calcium released by the bones leads to osteoporosis and osteomalacia (both bone-weakening diseases). Another result of hyperparathyroidism is kidney stones, because of high levels of calcium excreted into the urine by the kidneys. Hyperparathyroidism is quite rare in children.
What causes hyperparathyroidism?
Causes of hyperparathyroidism include benign (non-cancerous) tumors on the parathyroid glands or enlargement of the parathyroid glands.
What are symptoms of hyperparathyroidism?
According to a recent study, children with hyperparathyroidism experience more severe symptoms than adults. The following are the most common symptoms of hyperparathyroidism in children. However, each child may experience symptoms differently. Symptoms may include:
  • kidney pain (due to the presence of kidney stones)
  • diminished bone density that causes bone pain
  • aches and pains
  • abdominal pain
  • nausea
  • vomiting
  • fatigue
  • excessive urination
  • confusion
  • muscle weakness
The symptoms of hyperparathyroidism may resemble other conditions or medical problems. Always consult your child's physician for a diagnosis.

95. Hyperparathyroidism
hyperparathyroidism? Primary hyperparathyroidism is a disorder of theparathyroid glands. Most people What is hyperparathyroidism? If the
http://www.clevelandclinic.org/health/health-info/docs/1000/1012.asp?index=5848

96. Hyperparathyroidism - Mesa Veterinary Hospital
hyperparathyroidism by Erin O'Donnell, DVM. Primary hyperparathyroidismis caused by a tumor or overgrowth of the parathyroid gland.
http://www.mesavet.com/library/hyperparathyroidism.htm
Hyperparathyroidism
by Erin O'Donnell DVM The parathyroid gland helps regulate calcium levels in the blood. When calcium levels are low, the parathyroid gland released parathyroid hormone (PTH), which causes the release of calcium from the bones into the blood. The parathyroid gland is found very close to the thyroid gland, in the neck region. Primary hyperparathyroidism is a condition in which the parathyroid gland secretes too much PTH, and causes there to be too much calcium in the blood. Primary hyperparathyroidism is caused by a tumor or overgrowth of the parathyroid gland. Typically the animals who get primary hyperparathyroidism are middle aged to older dogs and cats. Five out of the seven reported cases of the disease in cats were in female Siamese cats. The signs of hyperparathyroidism are usually related to hypercalcemia (too much calcium in the blood), but are somewhat vague. Decreased appetite, lethargy, and depression are the most common signs. Some animals will drink lots of water and have to urinate often. Less frequently, owners may notice constipation, weakness, shivering, twitching, or vomiting. Many times there are no signs, and the increased blood calcium is found when the veterinarian checks a blood chemistry panel for another problem. There are several things that must be done to diagnose primary hyperparathyroidism. Hyperparathyroidism can be secondary to kidney problems and nutritional deficiencies, and certain cancers can produce a PTH-like hormone that has the same effect as PTH. These are important things for the veterinarian to be able to eliminate as causes for elevated blood calcium. The veterinarian may also need to ultrasound the neck to look for a parathyroid tumor.

97. Welcome To ENH.org - Health Encyclopedia: Hyperparathyroidism
hyperparathyroidism. See the specific type of hyperparathyroidism. Expectations(prognosis) Varies depending on the specific type of hyperparathyroidism.
http://www.enh.org/Encyclopedia/ency/article/001215.asp

Disease Reference
Injury Reference Test Reference Nutrition Reference ... Symptoms Reference
Hyperparathyroidism
Disease Injury Nutrition Poison ... Z Definition: Excessive production of parathyroid hormone by the parathyroid glands.
Causes, incidence, and risk factors: Hyperparathyroidism can be caused by enlargement of one or more of the parathyroid glands or in response to abnormally low calcium levels in the blood.
The parathyroid glands are located at the front and base of the neck around the thyroid gland and produce parathyroid hormone . Parathyroid hormone regulates calcium, phosphorus, and magnesium balance within the blood and bone by maintaining a balance between the mineral levels in the blood and the bone. Excessive levels of parathyroid hormone disturbs this balance and results in increasing the blood calcium level by reabsorbing it from the bone and increasing absorption from the kidneys and intestines.
The effects of abnormal calcium levels are seen in several body systems including the skeletal, gastrointestinal, renal , muscular, and central nervous system
Related topics:
Symptoms:
Signs and tests:

98. Health Library Find Information On Hyperparathyroidism At
Find information on hyperparathyroidism at MerckSource. hyperparathyroidism. DefinitionExcessive production of parathyroid hormone by the parathyroid glands.
http://www.mercksource.com/pp/us/cns/cns_hl_adam.jspzQzpgzEzzSzppdocszSzuszSzcns

99. ENLmedical.com: Conditions And Concerns: Medical Encyclopedia: Hyperparathyroidi
Table of content. hyperparathyroidism. See the specific type of hyperparathyroidism.Prognosis Varies depending on the specific type of hyperparathyroidism.
http://www.enlmedical.com/article/001215.htm

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Hyperparathyroidism
Causes and Risks:
Hyperparathyroidism can be caused by enlargement of one or more of the glands or in response to an abnormally low calcium level in the blood.
The parathyroid glands are located at the front and base of the neck around the thyroid gland and produce parathyroid hormone . Parathyroid hormone regulates calcium, phosphorus, and magnesium balance within the blood and bone by maintaining a balance between the mineral levels in the blood and the bone. Excessive levels of parathyroid hormone disturbs this balance and results in increasing the blood calcium level by reabsorbing it from the bone and increasing absorption from the kidneys and intestines.
The effects of abnormal calcium levels are seen in several body systems including the skeletal, gastrointestinal, renal , muscular, and central nervous system
Related topics:
Prevention:
Maintaining an adequate intake of calcium may reduce risk.

100. ATE Responses
SUBJ (10/02) hyperparathyroidism. In what ways does hyperparathyroidismmimic ALS? REPLY from MDA Rup Tandan, MD, FRCP, MDA Clinic
http://www.mdausa.org/experts/question.cfm?id=2964

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