Akinetic Mutism - Medical Dictionary Of Popular Medical Terms To Help You Better An article about akinetic mutism, what it is, the cause and symptoms.Category Health Conditions and Diseases akinetic mutism akinetic mutism A state in which a person is unspeaking (mute) andunmoving (akinetic). A textbook on clinical neurology observes http://www.medterms.com/script/main/art.asp?articlekey=6990
Meaning Of Akinetic Mutism Home World News. US People Word Wise Science Math Sports Cool StuffGames and Quizzes Homework Center Funbrain, dictionaryDictionary http://www.factmonster.com/ipd/A0311365.html
Member Sign In A 76year-old man developed akinetic mutism after 3 days of receivinglow-dosage baclofen. Focus On http://www.medscape.com/viewarticle/417965
WorldMedicus A description of akinetic mutism, with synonyms, related subjects and a link to reviews and editorials. http://www.worldmedicus.com/servlet/Controller/$7006041601950000.sj/
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Extractions: Search through: Entire site Akinetic Mutism DIRECTORY Health/Fitness Diseases Neurological Disorders ... Medicine Net - An article about akinetic mutism, what it is, the cause and symptoms. WorldMedicus - A description of akinetic mutism, with synonyms, related subjects and a link to reviews and editorials.
Akinetic Mutism - General Practice Notebook medical information from General Practice Notebook. akinetic mutism.akinetic mutism is a variety of stupor in which the patient is http://www.gpnotebook.co.uk/cache/483393567.htm
Extractions: akinetic mutism Akinetic mutism is a variety of stupor in which the patient is unable to talk or carry out purposeful behaviour but may lie with eyes open, seemingly unaware of what is going on around him. It results from bilateral damage to the orbital surface of the frontal lobes. The patient appears awake and has normal ocular movement but does not speak, is incontinent, and has minimal motor response to painful stimulation. It is seen in severe anterior cerebral artery strokes and less frequently, in hydrocephalus.
A Case Of Unpaired Anterior Cerebral Artery Occlusion Producing Akinetic Mutism Volume 47 Issue 2 1999 LETTERS TO EDITOR VOL472 A Case of Unpaired Anterior Cerebral Artery Occlusion Producing akinetic mutism A. Oommen, M. Madhusudanan Department of Medicine, Medical College Kottayam, Kerala, India. ACA occlusion by an embolus from the heart after a silent myocardial infarction producing akinetic mutism is presented. http://www.neurologyindia.com/vol47-2/1136fl.shtml
Extractions: Aplasia or congenital absence of one anterior cerebral artery (ACA) is seen in only 1% of angiograms. ACA territory infarcts constitute only 0.6% to 3% of all acute ischaemic strokes. Bilateral infarcts due to unpaired ACA occlusion is still more rare. A case of unpaired ACA occlusion by an embolus from the heart after a silent myocardial infarction producing akinetic mutism is presented. A 49 year old former airman was admitted with history of sudden onset of loss of consciousness and right focal seizures becoming generalized. He had exertional breathlessness for two days prior to admission. He was a heavy smoker and alcoholic. On admission, he was unconscious but his vital signs were stable. Ocular fundi were normal. He was moving all the four limbs equally. Plantars were equivocal. There was a loud fourth heart sound but no cardiac enlargement or murmurs. Seizures were controlled with intravenous phenytoin and diazepam. The next day, the patient was noticed to be more alert. He was opening his eyes, had spontaneous blinking and was following the examiner with his eyes. He was however mute and incontinent. All deep tendon jerks were brisk. Gegenhalten and grasp reflex were present bilaterally. Sensations could not be tested.
Assc-seminar-impaired.txt and coma, vegetative states, akinetic mutism, absence and partial complex seizures, hyperkinetic mutism, and others. http://tvisnius.ucs.indiana.edu/tom/assc/assc-seminar-impaired.txt
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Ask Jeeves: Search Results For "Akinetic" She has been in a comalike state known as akinetic mutism for ten yearsand receives around the clock care from a staff of nurses http://webster.directhit.com/webster/search.aspx?qry=Akinetic
Coma And Other Disorders Of Consciousness - Introduction Cairns, H., RC Oldfield, JB Pennybacker And D. Whitteridge akinetic mutismwith an epidermoid cyst of the 3rd ventricle Brain 64 (1941) 273290. http://sommeil.univ-lyon1.fr/articles/jouvet/hcn_69/contents.html
Extractions: Handbook of Clinical Neurology Vol.3. P. J. Vinken and G. W. Bruyn , eds. North-Holland Publishing Company. Amsterdam,(1969) TABLE OF CONTENTS Introduction Physiopathological basis of coma (introductory remarks) Nervous structures necessary for consciousness Periodic physiological dissolution of consciousness: sleep and coma ... Tentative anatomoclinical classification FIGURES Select a figure Figure 1 Figure 2 Figure 3 Figure 4 Figure 5 Figure 6 Figure 7 Figure 8 Figure 9 MICHEL JOUVET Page of Michel Jouvet His articles on this server and of reactivity , will lead to another definition borrowed from psychophysiology. According to this definition, consciousness is that central nervous process which gives significance to a stimulus from the external environment. We can thus understand by consciousness that function of the nervous system which is concerned with the perceptual experience of information from the environment and from our own body (Alajouanine 1957). The multiple disintegration of consciousness observed in coma can therefore be defined as the absence, in the patient, of objective clinical (or paraclinical) signs of appreciation of his environment. It is this behavioural deficiency that the clinician explores by simple means: failure to rouse a comatose patient by calling his name or applying a painful stimulus.
Tentative Anatomoclinical Classification (1941) under the name of akinetic mutism. 16 (1964) 285289. Daly, DD And JG.Loveakinetic mutism (a case report) Neurology (Minneap.) 8 (1958) 238-260. http://sommeil.univ-lyon1.fr/articles/jouvet/hcn_69/p8.html
Extractions: Handbook of Clinical Neurology Vol.3. P. J. Vinken and G. W. Bruyn , eds. North-Holland Publishing Company. Amsterdam,(1969) TABLE OF CONTENTS Introduction Physiopathological basis of coma (introductory remarks) Nervous structures necessary for consciousness Periodic physiological dissolution of consciousness: sleep and coma ... Tentative anatomoclinical classification FIGURES Select a figure Figure 1 Figure 2 Figure 3 Figure 4 Figure 5 Figure 6 Figure 7 Figure 8 Figure 9 MICHEL JOUVET Page of Michel Jouvet His articles on this server By correlating the preceding classification with anatomical observations on our own cases and others described in the literature, we have been able to distinguish four main anatomoclinical stages in prolonged coma ( Fig.8 The reactive apathic hypoperceptive syndrome The reactive hyperpathic-hypertonic aperceptivity syndrome In all the cases, the lesion is similar. There is either massive cortical involvement (Denst et al. 1958) or diffuse degenerative changes in the white matter of the cerebral hemispheres. The brainstem is intact in the majority of cases (with the exception of the pyramidal fibres which frequently show signs of degeneration). The areactive apathic normotonic aperceptivity syndrome This group concerns cases of very deep coma in which survival is usually limited to a few weeks. Perceptivity is lost (P5). Nonspecific reactions are altered (R2-R3) as well as reaction to pain (D2-D3), but autonomic reactions are normal. In most cases, there is no definite hypertonicity. Patients described by Jefferson (1952) under the name of parasomnia, by French (1952) under the name of prolonged uncounsciousness, and by Cravioto et al. (1958), and Trillet (1961), present the same symptomatology. The lesion common to all these patients is one affecting the upper part of the brainstem (mesencephalic reticular formation), but in most cases, there were also associated lesions of the cortex or white matter. It appears that total or subtotal destruction of the reticular system is responsible for the more severe losses of reactivity.
Vol.8 - 3. Establishment Of The CJD Surveillance Unit 4.4/1.11.3; S50 Allen paras 13-14 3 Myoclonus - muscle spasms 4 akinetic mutism- impairment of voluntary muscle movement, including the loss of speech 5 EEG http://www.bseinquiry.gov.uk/report/volume8/chapterc.htm
Extractions: The work of the CJDSU was reviewed by the MRC Allen Committee, chaired by Professor Ingrid Allen, which met four times between 1991 and 1995. This Committee was established on the basis of concerns raised by Professor Allen at a meeting of the MRC Coordinating Committee for Research on Spongiform Encephalopathies in Man (The Murray Committee; see vol. 2: Science ) in October 1990. She considered that the design of Dr Will's study could mean that atypical features of dementia, like those exhibited by patients developing CJD as a result of treatment with human growth hormone, might fall outside the study. A Clinical Subcommittee was therefore established to coordinate and facilitate studies relating to the epidemiological monitoring and neuropathological definition of human SEs, to monitor patients at risk of iatrogenic infection and to apply new methodologies and technologies to refine the definition and improve diagnosis of SEs. The Allen Committee had powers to co-opt relevant experts when necessary. Dr Will was a co-opted member of the group.
Vol.8 - 6. Diagnosis, Treatment And Care Of VCJD Patients Next Return to top of page 1 IBD3 tab 9 2 Dysaesthesiae unpleasant abnormalsensations 3 akinetic mutism - paralysis and inability to speak 4 IBD3 tab 9. http://www.bseinquiry.gov.uk/report/volume8/chaptee2.htm
Extractions: On 21 March 1996, the CJDSU circulated to all neurologists in the UK a description of the clinical and pathological features of the new phenotype. The clinical features of the new phenotype of CJD described in the circular were as follows: An early age of onset or death (average 27.6 years, range 18-41 years). A prolonged duration of illness (average 13.1 months, range 7.5-24 months). A predominantly psychiatric presentation including anxiety, depression, withdrawal and progressive behavioural changes. The first evidence of neurological involvement in four patients was dysaesthesiae in the limbs and/or face. Development of a cerebellar syndrome with problems with gait and limb muscle coordination after a period of weeks or months. Development of forgetfulness and memory disturbance, often late in the clinical course, which progressed to severe cognitive impairment and a state of akinetic mutism
02-13 and empirically based taxonomy for these primary disorders of consciousness, involvingcoma, persistent vegetative state, akinetic mutism, hyperkinetic mutism http://www.imprint-academic.demon.co.uk/T2000/02-13.html
Extractions: With the exception of work by Panksepp, Newman and Baars, and Damasio, there has been virtually no systematic work in consciousness theory on systems beneath the thalamus in terms of their critical participation in foundations for consciousness, although virtually everyone agrees that numerous reticular systems, particularly the core monoamine nuclei, are essential for consciousness. However, beyond notions of the ‘non-specific arousal’ functions supported in these core nuclei, the ventral brain gets very little attention and consciousness theory outside of the above noted authors. This talk will outline the columnar architecture and connectivities of PAG, its role in the organizing prototypes states of emotion, and the re-entry of PAG with the extended reticular thalamic activating system (ERTAS). At the end we will outline some potential implications of these connectivities for possible functional correlates of PAG networks that are just starting to be mapped. Over all, we will look at many lines of evidence that PAG should be conceptualized as a peri-reticular structure that has a foundational role in emotion, and in generating the meaningful organization of behavior by the brain through prototype emotional states. Cerebral Organization of Self, Two Types of Emotions and Consciousness
Searchalot Directory For Akinetic Mutism Related Web Sites. Medicine Net An article about akinetic mutism,what it is, the cause and symptoms. WorldMedicus - A description http://www.searchalot.com/Top/Health/ConditionsandDiseases/NeurologicalDisorders
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